The 13 protein coding genes and 24 RNA only coding genes on the mitochondria (source: https://www.tandfonline.com/doi/full/10.1080/23802359.2019.1693307) did not come from commercial stock in the situation you mentioned.  Those genes tell you nothing about ancestry other than the wild maternal line is unbroken.  Those 13 protein coding genes are probably 100% ineffective at influencing livability as that is the normal case.  Any mutation in a coding region usually causes death of that mitochondria.  The variations in mitochondria are nearly 100% contained in the non coding regions so those variations would very seldom express in any way in the phenotype.  If the non coding regions did effect phenotype it would be via changing the rate at which one of the coding genes expressed and this also would often result in mitochondrial death.

Knowing the mitochondrial linage tells you nothing at all about the actual genetic lineage that determines phenotype of the animal.  The fact is you could put any mitochondria you wished into a pure Italian or a pure Carny if such pure stock exists someplace in the world.

To illustrate how conserved the genes in mitochondria are humans and honey bees have exactly the same number of protein and RNA only genes in their mitochondria.

Dick

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