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Sun, 10 Feb 2008 02:45:00 -0500 |
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I had posted on this January 3rd 2008: Adipose tissue hangs on to
progesterone from this study I read: This is just an excerpt.
The population subgroups with the highest proportion of
overweight and obese women often are characterized by the lowest rates of
initiation and shortest durations of breastfeeding. We previously documented
that these 2 population-level trends may be related. In a population of white
women who lived in a rural area, we observed that prepregnant overweight
and obesity were associated with failure to initiate and also to sustain
lactation. The means by which being overweight or obese negatively affect
lactational performance is unknown and likely to be multifactorial in origin,
including the simple mechanical difficulties of latching on and proper
positioning of the infant. In addition, we have shown that prepregnant body
mass index (BMI) is negatively associated with the timing of lactogenesis II,
the onset of copious milk secretion. Although the effects of obesity on the
prolactin response to infant suckling have never been studied, we postulated
that maternal obesity could compromise this important response. We proposed
that this might occur because obesity alters the 24-hour spontaneous release
of prolactin and also because prolactin secretion is blunted in response to
various stimuli among obese subjects. The fall in progesterone concentration
that occurs immediately postpartum is the trigger for the onset of copious milk
secretion, but maintenance of prolactin and cortisol concentrations is
necessary for this trigger to be effective. Adipose tissue concentrates
progesterone. We proposed that this additional source of progesterone would
lead to consistently higher progesterone concentrations among obese
compared with normal-weight women. This, in turn, would lead to a delay in
reaching the appropriate concentration to trigger the onset of lactogenesis II.
This wasn't the best study in the world, but does tend to indicate if true that
adipose tissue holds onto progesterone. The progesterone then has an effect
on prolactin and lactogenesis II . Maybe one reason slender women have
a "normal" supply of milk, but we are mistaking it for an overabundant supply?
Just a thought.
robin
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